Cutaneous deposition diseases. Part I
Identifieur interne : 002829 ( Main/Exploration ); précédent : 002828; suivant : 002830Cutaneous deposition diseases. Part I
Auteurs : Diane M. Touart ; Purnima SauSource :
- Journal of the American Academy of Dermatology [ 0190-9622 ] ; 1998.
English descriptors
- Teeft :
- Abnormality, Acad, Acad dermatol, Acute attacks, American academy, Amyloid, Amyloid deposits, Amyloid filaments, Amyloidosis, Anecdotal reports, Arch dermatol, Autosomal, Basal, Blood vessels, Bulla, Bullous, Clin, Clinical features, Clinical types, Collagen, Colloid, Colloid bodies, Colloid milium, Congenital erythropoietic porphyria, Copro, Coproporphyrin, Cutan, Cutanea, Cutaneous, Cutaneous amyloidosis, Cutaneous deposition disorders, Cutis, Degeneration, Deposit, Deposition, Dermal, Dermatol, Dermatol clin, Dermatology, Dermatology volume, Dermis, Dermoepidermal junction, Dimethyl sulfoxide, Direct immunofluorescence, Dmso, Eccrine glands, Elastic fibers, Enzyme deficiency, Eosinophilic, Epidermal, Epidermal basement membrane, Erythrocyte, Erythropoietic, Erythropoietic protoporphyria, Familial mediterranean fever, Fibril, Fibroblast, Filament, Hashimoto, Heme, Heme synthesis, Hemodialysis, Hereditary coproporphyria, High levels, Histologic, Human immunodeficiency virus infection, Hyalinosis, Hyalinosis cutis, Immunofluorescence, Immunoglobulin, Immunoglobulin light chains, Juvenile colloid milium, Lamina, Lesion, Lichen, Lichen amyloidosis, Lichen amyloidosus, Lipid, Lipoid, Lipoid proteinosis, Liver disease, Localized, Localized cutaneous amyloidosis, Macular, Macular amyloidosis, Milium, Multiple myeloma, Nodular, Nodular amyloidosis, Nodular colloid degeneration, Nodular cutaneous amyloidosis, Nodule, Original magnification, Papillary, Papillary dermis, Papule, Pathogenesis, Pathol, Perivascular, Plasma cells, Porphyria, Porphyria cutanea tarda, Porphyrin, Porphyrin metabolism, Precursor, Primary cutaneous amyloidosis, Proteinosis, Proto, Protoporphyria, Pseudoporphyria, Secondary amyloidosis, Skin fragility, Skin lesions, Solar elastosis, Subcutaneous, Systemic, Systemic amyloidosis, Systemic disease, Tarda, Touart, Ultrastructural, Ultrastructural features, Ultrastructural study, Urine, Uroporphyrin, Variegate porphyria, Vessel walls, Walter reed army.
Abstract
Abstract: The cutaneous deposition disorders are a group of unrelated conditions characterized by the presence of either endogenous or exogenous substances within the dermis or the subcutis. Part I of this two-part series will focus on metabolic processes involved in the endogenous deposition in the various forms of amyloidosis, porphyria, colloid milium, and lipoid proteinosis. We will also review the clinical, histologic, biochemical, and ultrastructural findings relevant to each disorder. Basic mechanisms of pathogenesis, diagnostic modalities, and treatment options are also discussed. (J Am Acad Dermatol 1998;39:149-71.) Learning Objective: After reading this article, participants should be familiar with the deposition disorders. On the basis of the clinical and histologic findings discussed for each disorder, clinicians should be able to distinguish between these often-confusing entities.
Url:
DOI: 10.1016/S0190-9622(98)70069-6
Affiliations:
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Part I</title><author><name sortKey="Touart, Diane M" sort="Touart, Diane M" uniqKey="Touart D" first="Diane M." last="Touart">Diane M. Touart</name><affiliation><wicri:noCountry code="subField">Center</wicri:noCountry></affiliation></author><author><name sortKey="Sau, Purnima" sort="Sau, Purnima" uniqKey="Sau P" first="Purnima" last="Sau">Purnima Sau</name><affiliation><wicri:noCountry code="subField">Center</wicri:noCountry></affiliation></author></analytic><monogr></monogr><series><title level="j">Journal of the American Academy of Dermatology</title><title level="j" type="abbrev">YMJD</title><idno type="ISSN">0190-9622</idno><imprint><publisher>ELSEVIER</publisher><date type="published" when="1998">1998</date><biblScope unit="volume">39</biblScope><biblScope unit="issue">2</biblScope><biblScope unit="page" from="149">149</biblScope><biblScope unit="page" to="171">171</biblScope></imprint><idno type="ISSN">0190-9622</idno></series></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">0190-9622</idno></seriesStmt></fileDesc><profileDesc><textClass><keywords scheme="Teeft" xml:lang="en"><term>Abnormality</term><term>Acad</term><term>Acad dermatol</term><term>Acute attacks</term><term>American academy</term><term>Amyloid</term><term>Amyloid deposits</term><term>Amyloid filaments</term><term>Amyloidosis</term><term>Anecdotal reports</term><term>Arch dermatol</term><term>Autosomal</term><term>Basal</term><term>Blood vessels</term><term>Bulla</term><term>Bullous</term><term>Clin</term><term>Clinical features</term><term>Clinical types</term><term>Collagen</term><term>Colloid</term><term>Colloid bodies</term><term>Colloid milium</term><term>Congenital erythropoietic porphyria</term><term>Copro</term><term>Coproporphyrin</term><term>Cutan</term><term>Cutanea</term><term>Cutaneous</term><term>Cutaneous amyloidosis</term><term>Cutaneous deposition disorders</term><term>Cutis</term><term>Degeneration</term><term>Deposit</term><term>Deposition</term><term>Dermal</term><term>Dermatol</term><term>Dermatol clin</term><term>Dermatology</term><term>Dermatology volume</term><term>Dermis</term><term>Dermoepidermal junction</term><term>Dimethyl sulfoxide</term><term>Direct immunofluorescence</term><term>Dmso</term><term>Eccrine glands</term><term>Elastic fibers</term><term>Enzyme deficiency</term><term>Eosinophilic</term><term>Epidermal</term><term>Epidermal basement membrane</term><term>Erythrocyte</term><term>Erythropoietic</term><term>Erythropoietic protoporphyria</term><term>Familial mediterranean fever</term><term>Fibril</term><term>Fibroblast</term><term>Filament</term><term>Hashimoto</term><term>Heme</term><term>Heme synthesis</term><term>Hemodialysis</term><term>Hereditary coproporphyria</term><term>High levels</term><term>Histologic</term><term>Human immunodeficiency virus infection</term><term>Hyalinosis</term><term>Hyalinosis cutis</term><term>Immunofluorescence</term><term>Immunoglobulin</term><term>Immunoglobulin light chains</term><term>Juvenile colloid milium</term><term>Lamina</term><term>Lesion</term><term>Lichen</term><term>Lichen amyloidosis</term><term>Lichen amyloidosus</term><term>Lipid</term><term>Lipoid</term><term>Lipoid proteinosis</term><term>Liver disease</term><term>Localized</term><term>Localized cutaneous amyloidosis</term><term>Macular</term><term>Macular amyloidosis</term><term>Milium</term><term>Multiple myeloma</term><term>Nodular</term><term>Nodular amyloidosis</term><term>Nodular colloid degeneration</term><term>Nodular cutaneous amyloidosis</term><term>Nodule</term><term>Original magnification</term><term>Papillary</term><term>Papillary dermis</term><term>Papule</term><term>Pathogenesis</term><term>Pathol</term><term>Perivascular</term><term>Plasma cells</term><term>Porphyria</term><term>Porphyria cutanea tarda</term><term>Porphyrin</term><term>Porphyrin metabolism</term><term>Precursor</term><term>Primary cutaneous amyloidosis</term><term>Proteinosis</term><term>Proto</term><term>Protoporphyria</term><term>Pseudoporphyria</term><term>Secondary amyloidosis</term><term>Skin fragility</term><term>Skin lesions</term><term>Solar elastosis</term><term>Subcutaneous</term><term>Systemic</term><term>Systemic amyloidosis</term><term>Systemic disease</term><term>Tarda</term><term>Touart</term><term>Ultrastructural</term><term>Ultrastructural features</term><term>Ultrastructural study</term><term>Urine</term><term>Uroporphyrin</term><term>Variegate porphyria</term><term>Vessel walls</term><term>Walter reed army</term></keywords></textClass><langUsage><language ident="en">en</language></langUsage></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">Abstract: The cutaneous deposition disorders are a group of unrelated conditions characterized by the presence of either endogenous or exogenous substances within the dermis or the subcutis. Part I of this two-part series will focus on metabolic processes involved in the endogenous deposition in the various forms of amyloidosis, porphyria, colloid milium, and lipoid proteinosis. We will also review the clinical, histologic, biochemical, and ultrastructural findings relevant to each disorder. Basic mechanisms of pathogenesis, diagnostic modalities, and treatment options are also discussed. (J Am Acad Dermatol 1998;39:149-71.) Learning Objective: After reading this article, participants should be familiar with the deposition disorders. On the basis of the clinical and histologic findings discussed for each disorder, clinicians should be able to distinguish between these often-confusing entities.</div></front></TEI><affiliations><list></list><tree><noCountry><name sortKey="Sau, Purnima" sort="Sau, Purnima" uniqKey="Sau P" first="Purnima" last="Sau">Purnima Sau</name><name sortKey="Touart, Diane M" sort="Touart, Diane M" uniqKey="Touart D" first="Diane M." last="Touart">Diane M. Touart</name></noCountry></tree></affiliations></record>Pour manipuler ce document sous Unix (Dilib)
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